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The commonly accepted pathogenetic theory is herein reported, from endothelial injury with deposition of noninfective sterile thrombotic vegetations to transient bacteremia with microorganism adhesion (injury-thrombus-infection theory).
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Adherence of bacteria to heart valves in vitro. Infective endocarditis (IE) is an uncommon infection, occurring as a complication in varying percentages of bacteremic episodes. This review addresses the pathology of native valve endocarditis, including local (valvular and perivalvular destruction) and distal (embolism, metastatic infection, and septicemia) complications.
Infective endocarditis due to unusual or fastidious microorganisms. Unable to load your delegates due to an error Epub 2019 Oct 25.Buschmann K, Chaban R, Emrich AL, Youssef M, Kornberger A, Beiras-Fernandez A, Vahl CF.Innov Surg Sci. Intracardiac hemodynamics predisposing to endocarditis Berbari EF, Cockerill FR, Steckelberg JM. Clinical practice. Subscription will auto renew annually.Chang FY et al. Elsevier Science The characteristic lesions consist of vegetations composed of platelets, fibrin, microorganisms, and inflammatory cells, as well as leaflet disruption. Infective endocarditis is a very complex disease with a serious prognosis.
Glucosyltransferases of viridans group streptococci modulate interleukin-6 and adhesion molecule expression in endothelial cells and augment monocytic cell adherence.
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